Abrahams, S., Haylett, W. L., Johnson, G., Carr, J. A., & Bardien, S. (2019). Antioxidant effects of curcumin in models of neurodegeneration, aging, oxidative and nitrosative stress: A review. Neuroscience, 406, 1-21.
Andrukhov, O., Haririan, H., Bertl, K., Rausch, W. D., Bantleon, H. P., Moritz, A., & Rausch‐Fan, X. (2013). Nitric oxide production, systemic inflammation and lipid metabolism in periodontitis patients: possible gender aspect. Journal of Clinical Periodontology, 40(10), 916-923.
Arnér, E. S., & Holmgren, A. (2000). Physiological functions of thioredoxin and thioredoxin reductase. European Journal of Biochemistry, 267(20), 6102-6109.
Baek, S. S. (2016). Role of exercise on the brain. Journal of Exercise Rehabilitation, 12(5), 380-385.
Biswas, J., Roy, S., Mukherjee, S., Sinha, D., & Roy, M. (2010). Indian spice curcumin may be an effective strategy to combat the genotoxicity of arsenic in Swiss albino mice. Asian Pacific Journal of Cancer Prevention, 11(1), 239-47.
Brinkley, T. E., Fenty-Stewart, N. M., Park, J. Y., Brown, M. D., & Hagberg, J. M. (2009). Plasma nitrate/nitrite levels are unchanged after long-term aerobic exercise training in older adults. Nitric Oxide, 21(3-4), 234-238.
Chan, M. M. Y., Ho, C. T., & Huang, H. I. (1995). Effects of three dietary phytochemicals from tea, rosemary and turmeric on inflammation-induced nitrite production. Cancer Letters, 96(1), 23-29.
D’amelio, M., Cavallucci, V., & Cecconi, F. (2010). Neuronal caspase-3 signaling: not only cell death. Cell Death and Differentiation, 17(7), 1104-1114.
Farkhondeh, T., Samarghandian, S., & Samini, F. (2016). Antidotal effects of curcumin against neurotoxic agents: An updated review. Asian Pacific Journal of Tropical Medicine, 9(10), 947-953.
Flora, S. J., Mittal, M., Pachauri, V., & Dwivedi, N. (2012). A possible mechanism for combined arsenic and fluoride induced cellular and DNA damage in mice. Metallomics, 4(1), 78-90.
Freitas, D. A., Rocha-Vieira, E., Soares, B. A., Nonato, L. F., Fonseca, S. R., Martins, J. B., ... & Meeusen, R. (2018). High intensity interval training modulates hippocampal oxidative stress, BDNF and inflammatory mediators in rats. Physiology and Behavior, 184, 6-11.
Freitas, R. M., Vasconcelos, S. M., Souza, F. C., Viana, G. S., & Fonteles, M. M. (2005). Oxidative stress in the hippocampus after pilocarpine‐induced status epilepticus in Wistar rats. The FEBS Journal, 272(6), 1307-1312.
Gamble, M. V., Liu, X., Ahsan, H., Pilsner, J. R., Ilievski, V., Slavkovich, V., ... & Graziano, J. H. (2005). Folate, homocysteine, and arsenic metabolism in arsenic-exposed individuals in Bangladesh. Environmental Health Perspectives, 113(12), 1683-1688.
Hafstad, A. D., Lund, J., Hadler-Olsen, E., Höper, A. C., Larsen, T. S., & Aasum, E. (2013). High-and moderate-intensity training normalizes ventricular function and mechanoenergetics in mice with diet-induced obesity. Diabetes, 62(7), 2287-2294.
Høydal, M. A., Wisløff, U., Kemi, O. J., & Ellingsen, Ø. (2007). Running speed and maximal oxygen uptake in rats and mice: practical implications for exercise training. European Journal of Cardiovascular Prevention and Rehabilitation, 14(6), 753-760.
Hosseinzadeh, S., Roshan, V. D., & Mahjoub, S. (2013). Continuous exercise training and curcumin attenuate changes in brain-derived neurotrophic factor and oxidative stress induced by lead acetate in the hippocampus of male rats. Pharmaceutical Biology, 51(2), 240-245.
Ishrat, T., Hoda, M. N., Khan, M. B., Yousuf, S., Ahmad, M., Khan, M. M., ... & Islam, F. (2009). Amelioration of cognitive deficits and neurodegeneration by curcumin in rat model of sporadic dementia of Alzheimer’s type (SDAT). European Neuropsychopharmacology, 19(9), 636-647.
Kantari, C., & Walczak, H. (2011). Caspase-8 and bid: caught in the act between death receptors and mitochondria. Biochimica et Biophysica Acta (BBA)-Molecular Cell Research, 1813(4), 558-563.
Li, W., Suwanwela, N. C., & Patumraj, S. (2016). Curcumin by down-regulating NF-kB and elevating Nrf2, reduces brain edema and neurological dysfunction after cerebral I/R. Microvascular Research, 106, 117-127.
Liguori, I., Russo, G., Curcio, F., Bulli, G., Aran, L., Della-Morte, D., ... & Abete, P. (2018). Oxidative stress, aging, and diseases. Clinical Interventions in Aging, 13, 757.
Liou, C. M., Tsai, S. C., Kuo, C. H., Ting, H., & Lee, S. D. (2014). Cardiac Fas-dependent and mitochondria-dependent apoptosis after chronic cocaine abuse. International Journal of Molecular Sciences, 15(4), 5988-6001.
Mai, Z., & Liu, H. (2009). Boolean network-based analysis of the apoptosis network: irreversible apoptosis and stable surviving. Journal of Theoretical Biology, 259(4), 760-769.
Obeid, R., & Herrmann, W. (2006). Mechanisms of homocysteine neurotoxicity in neurodegenerative diseases with special reference to dementia. Federation of the European Biochemical Societies, 580(13), 2994-3005.
Park, Y. J., Ko, J. W., Jang, Y., & Kwon, Y. H. (2013). Activation of AMP-activated protein kinase alleviates homocysteine- mediated neurotoxicity in SH-SY5Y cells. Neurochemical Research, 38(8), 1561-1571.
Phunchago, N., Wattanathorn, J., & Chaisiwamongkol, K. (2015). Tiliacora triandra, an anti-intoxication plant, improves memory impairment, neurodegeneration, cholinergic function, and oxidative stress in hippocampus of ethanol dependence rats. Oxidative Medicine and Cellular Longevity, 918426.
Prakash, C., & Kumar, V. (2016). Arsenic-induced mitochondrial oxidative damage is mediated by decreased PGC-1α expression and its downstream targets in rat brain. Chemico-Biological Interactions, 256, 228-235.
Prasad, P., & Sinha, D. (2017). Low-level arsenic causes chronic inflammation and suppresses expression of phagocytic receptors. Environmental Science and Pollution Research, 24(12), 11708-11721.
Ringman, J. M., Frautschy, S. A., Teng, E., Begum, A. N., Bardens, J., Beigi, M., ... & Porter, V. (2012). Oral curcumin for Alzheimer’s disease: tolerability and efficacy in a 24-week randomized, double blind, placebo-controlled study. Alzheimer’s Research and Therapy, 4(5), 43.
Sachdev, P. S. (2005). Homocysteine and brain atrophy. Progress in Neuro-Psychopharmacology and Biological Psychiatry, 29(7), 1152-1161.
Selvaraj, V., Armistead, M. Y., Cohenford, M., & Murray, E. (2013). Arsenic trioxide (As2O3) induces apoptosis and necrosis mediated cell death through mitochondrial membrane potential damage and elevated production of reactive oxygen species in PLHC-1 fish cell line. Chemosphere, 90(3), 1201-1209.
Shibata, M., Araki, N., Hamada, J., Sasaki, T., Shimazu, K., & Fukuuchi, Y. (1996). Brain nitrite production during global ischemia and reperfusion: an in vivo microdialysis study. Brain Research, 734(1-2), 86-90.
Shirvani, H., Aslani, J., Mohammadi, Z. F., & Arabzadeh, E. (2019). Short-term effect of low-, moderate-, and high-intensity exercise training on cerebral dopamine neurotrophic factor (CDNF) and oxidative stress biomarkers in brain male Wistar rats. Comparative Clinical Pathology, 28(2), 369-376.
Sun, H., Yang, Y., Shao, H., Sun, W., Gu, M., Wang, H., ... & Gao, Y. (2017). Sodium arsenite-induced learning and memory impairment is associated with endoplasmic reticulum stress-mediated apoptosis in rat hippocampus. Frontiers in Molecular Neuroscience, 10, 286.
Svensson, M., Lexell, J., & Deierborg, T. (2015). Effects of physical exercise on neuroinflammation, neuroplasticity, neurodegeneration, and behavior: what we can learn from animal models in clinical settings. Neurorehabilitation and Neural Repair, 29(6), 577-589.
Tsai, C. L., Wang, C. H., Pan, C. Y., & Chen, F. C. (2015). The effects of long-term resistance exercise on the relationship between neurocognitive performance and GH, IGF-1, and homocysteine levels in the elderly. Frontiers In Behavioral Neuroscience, 9, 23.
Vincent, K. R., Braith, R. W., Bottiglieri, T., Vincent, H. K., & Lowenthal, D. T. (2003). Homocysteine and lipoprotein levels following resistance training in older adults. Preventive Cardiology, 6(4), 197-203.
Wagner, G., Herbsleb, M., Cruz, F. D. L., Schumann, A., Brünner, F., Schachtzabel, C., ... & Reichenbach, J. R. (2015). Hippocampal structure, metabolism, and inflammatory response after a 6-week intense aerobic exercise in healthy young adults: a controlled trial. Journal of Cerebral Blood Flow & Metabolism, 35(10), 1570-1578.
Wang, X., Mandal, A. K., Saito, H., Pulliam, J. F., Lee, E. Y., Ke, Z. J., ... & Tucker, T. (2012). Arsenic and chromium in drinking water promote tumorigenesis in a mouse colitis-associated colorectal cancer model and the potential mechanism is ROS-mediated Wnt/β-catenin signaling pathway. Toxicology and Applied Pharmacology, 262(1), 11-21.
Wolfe, M. S. (Ed.). (2018). The Molecular and Cellular Basis of Neurodegenerative Diseases: Underlying Mechanisms. 1st Edition, Kindle Edition, Academic Press.